


Nindanib 150 mg Capsule | 10 Capsules in 1 Strip

- Manufacturer/Marketed By
Glenmark Pharmaceuticals Ltd
- Active Pharmaceutical Ingredient
Nintedanib 150 mg
(Inclusive of all taxes)
Introduction of Nindanib 150 mg Capsule
Nindanib 150 mg Capsule is a targeted cancer drug. It is used to treat pulmonary fibrosis in which the cause is not known ( idiopathic pulmonary fibrosis). The chemical formula of this drug is C31H33N5O4. Nindanib 150 mg Capsule is classified as a kinase inhibitor. Nindanib 150 mg Capsule blocks the action of enzymes that cause fibrosis. It is available as an oral capsule form easily taken by mouth.
Uses of Nindanib 150 mg Capsule
- Idiopathic pulmonary fibrosis
- Impaired respiratory function
- Interstitial lung disease
- NSCLS ( Non small cell lung Cancer)
Benefits of Nindanib 150 mg Capsule
Nindanib 150 mg Capsule slows disease progression by reducing the rate of decline in forced vital capacity in those patients who have idiopathic pulmonary fibrosis with mild or moderate lung function impairment.
Side Effects of Nindanib 150 mg Capsule
- Loss of appetite
- Weight loss
- Fatigue
- Headache
- Unhealed wound
Most Common Side Effects of Nindanib 150 mg Capsule
- Nausea
- Vomiting
- Diarrhoea
- Stomach upset
Common Side Effects of Nindanib 150 mg Capsule
- Altered blood pressure
- Liver problems (jaundice)
- Dark brown urine
- Yellowing of the skin
- Lack of energy
- Shortness of breath
- Numbness or weakness
- Difficulty in speech
- Malena ( blood in stool)
How to Use Nindanib 150 mg Capsule?
Nindanib 150 mg Capsule is available in oral capsule form taken by mouth as directed by your doctor. Take it regularly as advised, frequency and dosage of the medicine will be decided by your doctor as per the severity of disease and certain other factors and how your body responds to the treatment.
How Nindanib 150 mg Capsule Works?
Nindanib 150 mg Capsule binds to fibroblast growth factor receptor's intracellular ATP binding pocket, PDGFR (platelet derived growth factor receptor) and VEGFRs ( Vascular growth factor receptors resulting in blockage of the receptors and the downstream signalling cascades.
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